Dherange, Parinita
Lang, Joshua
Qian, Pierre
Oberfeld, Blake
Sauer, William H.
Koplan, Bruce
Tedrow, Usha
2020-09-01T20:44:48Z
2020-09-01T20:44:48Z
2020
S2405-500X
https://doi.org/10.1016/j.jacep.2020.08.002
http://hdl.handle.net/20.500.12010/12573
Current understanding of the impact of COVID-19 on arrhythmias continues to evolve as new
data emerges. Cardiac arrhythmias are more common in critically ill COVID-19 patients. The
potential mechanisms that could result in arrhythmogenesis among COVID-19 patients includes
hypoxia caused by direct viral tissue involvement of lungs, myocarditis, abnormal host immune
response, myocardial ischemia, myocardial strain, electrolyte derangements, intravascular
volume imbalances and drug sides effects. To manage these arrhythmias, it is imperative to
increase the awareness of potential drug-drug interactions, to monitor QTc prolongation while
receiving COVID therapy and provide special considerations for patients with inherited
arrhythmia syndromes. It is also crucial to minimize exposure to COVID-19 infection by
stratifying the need for intervention and using telemedicine. As COVID-19 infection continues to
prevail with a potential for future surges, more data is required to better understand
pathophysiology and to validate management strategies.
Keywords: COVID-19, SARS-CoV-2, Arrhythmias, QT prolongation, Torsades De Pointes,
Channelopathies
38 páginas
image/jepg
eng
JACC: Clinical Electrophysiology
reponame:Expeditio Repositorio Institucional UJTL
instname:Universidad de Bogotá Jorge Tadeo Lozano
COVID-19
SARS-CoV-2
Arrhythmias
QT prolongation
Torsades De Pointes
Channelopathies
Myocarditis
Arrhythmias and COVID-19: A review
Artículo
Síndrome respiratorio agudo grave
COVID-19
SARS-CoV-2
Coronavirus
info:eu-repo/semantics/embargoedAccess
info:eu-repo/semantics/acceptedVersion
Acceso restringido
https://doi.org/10.1016/j.jacep.2020.08.002
http://purl.org/coar/resource_type/c_2df8fbb1