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dc.creatorVen Eng, Vik
dc.creatorWemyss, Madeleine A.
dc.creatorPearson, Jaclyn S.
dc.date.accessioned2020-09-04T19:39:02Z
dc.date.available2020-09-04T19:39:02Z
dc.date.created2020
dc.identifier.issn1084-9521spa
dc.identifier.otherhttps://doi.org/10.1016/j.semcdb.2020.08.005spa
dc.identifier.urihttp://hdl.handle.net/20.500.12010/12730
dc.description.abstractReceptor Interacting Protein Kinases (RIPKs) are cellular signaling molecules that are critical for homeostatic signaling in both communicable and non-communicable disease processes. In particular, RIPK1, RIPK2, RIPK3 and RIPK7 have emerged as key mediators of intracellular signal transduction including inflammation, autophagy and programmed cell death, and are thus essential for the early control of many diverse pathogenic organisms. In this review, we discuss the role of each RIPK in host responses to bacterial and viral pathogens, with a focus on studies that have used pathogen infection models rather than artificial stimulation with purifiedspa
dc.format.extent19 páginasspa
dc.format.mimetypeapplication/pdfspa
dc.language.isoengspa
dc.publisherSeminars in Cell and Developmental Biologyspa
dc.sourcereponame:Expeditio Repositorio Institucional UJTLspa
dc.sourceinstname:Universidad de Bogotá Jorge Tadeo Lozanospa
dc.subjectRIP kinasespa
dc.subjectInflammationspa
dc.subjectCell deathspa
dc.subjectBacterial infectionspa
dc.subjectViral infectionspa
dc.subjectPathogenspa
dc.titleThe diverse roles of RIP kinases in host-pathogen interactionsspa
dc.type.localArtículospa
dc.subject.lembSíndrome respiratorio agudo gravespa
dc.subject.lembCOVID-19spa
dc.subject.lembSARS-CoV-2spa
dc.subject.lembCoronavirusspa
dc.rights.accessrightsinfo:eu-repo/semantics/embargoedAccessspa
dc.type.hasversioninfo:eu-repo/semantics/acceptedVersionspa
dc.rights.localAcceso restringidospa
dc.identifier.doihttps://doi.org/10.1016/j.semcdb.2020.08.005spa
dc.type.coarhttp://purl.org/coar/resource_type/c_2df8fbb1spa


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